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Pathophysiological and nutritional aspects in the etiology and management of gastroesophageal reflux disease | British Journal of Nutrition | Cambridge Core
[17] [18] It is thought that GliA, GtmA, and GliT provide mechanisms for self-protection against gliotoxin toxicity for the fungi that produce and excrete gliotoxin
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Not only did BSO increase PMN infiltrate in the lung, but it also diminished PMN infiltration in the site of infection, thus increasing bacterial growth
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