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Inhibition of the FSP1 pathway can weaken the protective effect of edaravone on TBI, suggesting that edaravone inhibits ferroptosis induced by injury in early TBI by activating FSP1 pathway
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Subsequent work from the same group examined matrix metalloproteinase-2 expression by cultured dermal fibroblasts, with the copper requirement explicitly demonstrated by Cu-depletion controls [4]
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