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This regulation adds to well-known settings of the sensing [glucose] range due to other factors, including the proper K m of rodent glucose transporter GLUT2/SLC2A2 (human GLUT1/SLC2A1), K m , and the lack of product inhibition for glucokinase, existing smooth fluxes of glycolysis that supply the Krebs cycle, followed by the efficient supply of substrates (NADH, succinate) for the respiratory chain and OXPHOS (227, 290)

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doi:10.1007/s00246-020-02449-1