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Sulfur partitioning from cysteine controls T cell proliferation and effector function: Cell
This hypothesis is supported by experimental data on the combined application of LQ and licochalcone B (Dong et al
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Other pharmacological avenues for increasing BDNF signaling include TrkB agonists, such as cyclic peptides and small molecule drugs, to provide direct stimulation of TrkB receptors that are independent of the many BDNF secretion deficits that exist in many genetic models
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