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Mitochondrial targeting of coenzyme Q10 in inflammatory bowel disease: mechanisms, challenges, and prospects for clinical application - ScienceDirect

In heterodimeric GST complexes such as those formed by the cytosolic mu and alpha classes, however, the cleft between the two subunits is home to an additional high-affinity nonsubstrate xenobiotic binding site, which may account for the enzymes' ability to form heterodimers

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PUFAs, due to the inherent instability of their bis-allylic hydrogen atoms, are highly vulnerable to oxidative damage and act as the primary substrates for ferroptotic signaling (69)