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As described above, the lack of GSH, although without effect on re-oxidation of roGFP2 in the periplasm, slightly shifted the periplasmic redox homeostasis to more reducing conditions, as seen in the lowered steady state redox potential of roGFP-iL in those cells (Fig
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Acknowledgements We thank all researchers and colleagues from Key Laboratory of Molecular Biology in Beijing Anding Hospital of Capital Medical University and Beijing Institute for Brain Disorders for their great support Ethics declarations Competing interests The authors declare that they have no conflict of interest
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