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Glutathione/pH-responsive copper-based nanoplatform for amplified chemodynamic therapy through synergistic cycling regeneration of reactive oxygen species and dual glutathione depletion - ScienceDirect
In an orthotopic xenograft mouse model of GBM, the GFR nanoplatforms preferentially accumulated in the tumor tissue inhibited GBM growth, and prolonged survival by inducing GPX4-mediated ferroptosis
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Where they diverge is what they are and where they sit on the regulatory ladder
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