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Collectively, these findings highlight the notable roles of the PI3K/Akt and MAPK signaling pathways in the pathogenesis of bone and joint comorbidities associated with estrogen deficiency, positioning these pathways as promising targets for clinical intervention in OP and OA (Table II)
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However, here, we give the concept of “Medicine Food Homology (MFH)” (139), which encompasses dietary-relevant structures that are postulated to have therapeutic effects in orthopedic diseases through the gut–bone axis
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