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Exposure to R-1507, a mAb directed against IGF-1R, downregulated the nucleotide excision repair pathway in human small cell lung cancer (SCLC) cell lines and induced chemo- and radio-sensitization to cisplatin/ionizing radiation in selected SCLC models [32]
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PTEN, which antagonizes PI3K/AKT signaling, also associates with PI(3)P-positive endosomes to terminate AKT activation 23
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